Smith-Lemli-Opitz syndrome

نویسندگان

  • ALAN E DONNENFELD
  • ELAINE H ZACKAI
  • DONNA M McDONALD
چکیده

Mayeda K, Weiss L, Lindahl R, Dully M. Localization of the human lactate dehydrogenase B gene on the short arm of chromosome 12. Ain J Humn Genet 1974:26:59-64. 2 Kivlin JD, Fineman RM, Williams MS. Phenotype variation in the del(12p) syndrome. Am J Med Genet 1985;22:769-79. 3 Magenis E, Brown M G. Chamberlin J. et al. Resolution of breakpoints in a complex rearrangement by use of multiple staining techniques: confirmation of suspected 12pl2.3 intraband by deletion dosage effect of LDHB. Am J Med Genet 1981;9:95-103. 4 Tenconi R, Baccichetti C. Anglani F, Pellegrino PA, Kaplan JC, Junien C. Partial deletion of the short arm of chromosome 12(pl 1 ;pl3): report of a case. Ann Genet (Paris) 1975;18:95-8.

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Identification of a novel DHCR7 mutation in a Korean patient with Smith-Lemli-Opitz syndrome.

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Smith-Lemli-Opitz syndrome is a rare syndrome presenting with multiple congenital anomalies/mental retardation associated with low plasma cholesterol levels. The spectrum of severity extends from prenatal death with holoprosencephaly or other lethal malformations, to patients with minimal physical abnormalities and normal intelligence or minimal intellectual impairment. Congenital heart defect ...

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Smith-Lemli-Opitz Syndrome: A Case with Annular Pancreas

Smith-Lemli-Opitz syndrome is an autosomal recessive disease of cholesterol metabolism. It is a multiple malformation syndrome with typical dysmorphic features such as bitemporal narrowing, ptosis, epicanthus, microcephaly, micrognathia, and cardiovascular, skeletal, urogenital, and gastrointestinal anomalies. This report presents a typical case of Smith-Lemli-Opitz syndrome with annular pancre...

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Smith-Lemli-Opitz syndrome. II: A disorder of the fetal adrenals?

Two cases of Smith-Lemli-Opitz syndrome type II are presented. During the late stages of both pregnancies maternal oestriol levels were unrecordable and there was evidence of suppression of maternal adrenal function. We speculate on the existence of a primary defect in the fetal adrenals.

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Maternal apo E genotype is a modifier of the Smith-Lemli-Opitz syndrome.

BACKGROUND Smith-Lemli-Opitz syndrome (MIM 270400) is an autosomal recessive malformation and mental retardation syndrome that ranges in clinical severity from minimal dysmorphism and mild mental retardation to severe congenital anomalies and intrauterine death. Smith-Lemli-Opitz syndrome is caused by mutations in the Delta7 sterol-reductase gene (DHCR7; EC 1.3.1.21), which impair endogenous ch...

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تاریخ انتشار 2004